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Disorder of reverse cholesterol transport is considered to be a vital part of the pathogenesis of atherosclerosis. A large number of data point out a role of ABCA1 and ABCG1 while in the regulation of cholesterol transportation from macrophages and HDL development. A minimize while in the expression and useful action of these transporters is affiliated with the formation of “foam cells” and the progression of atherosclerosis. Unsaturated fatty acids for example palmitoleic, oleic, and linoleic acids have already been demonstrated to lessen ABCA1 expression at transcriptional along with posttranscriptional stages, cutting down cholesterol efflux (134, 135). Eicosapentaenoic and oleic acids also diminished ABCA1 promoter exercise in macrophages. Unsaturated fatty acids also suppressed ABCG1 gene expression via a system that includes LXR/RXR binding to promoters (136).
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